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Energy Metabolism
Focal Cerebral Ischemia
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Penumbral tissue alkalosis in focal cerebral ischemia: Relationship to energy metabolism, blood flow, and steady potential
Penumbral tissue alkalosis in focal cerebral ischemia: Relationship to energy metabolism, blood flow, and steady potential,10.1002/1531-8249(200004)47
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Penumbral tissue alkalosis in focal cerebral ischemia: Relationship to energy metabolism, blood flow, and steady potential
(
Citations: 37
)
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Tobias Back
,
Mathias Hoehn
,
Günter Mies
,
Elmar Busch
,
Bernd Schmitz
,
Kanehisa Kohno
Journal:
Annals of Neurology - ANN NEUROL
, vol. 47, no. 4, pp. 485-492, 2000
DOI:
10.1002/1531-8249(200004)47:4<485::AID-ANA12>3.3.CO;2-#
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Citation Context
(8)
...Dohmen
et al
, 2008), and their observation after diverse brain insults has renewed interest in their pathophysiological significance (
...
Yoshimasa Takeda
,
et al.
Metabolic and perfusion responses to recurrent peri-infarct depolariza...
...39,
...
Ming-hua Li
,
et al.
Calcium-permeable ion channels involved in glutamate receptor-independ...
...Wang
et al
, 2007), neurodegenerative diseases (
...
Suresh L Mehta
,
et al.
Neuroprotective role of mitochondrial uncoupling protein 2 in cerebral...
...AEP Mediates Stroke-Provoked SET Degradation and DNA Damage Stroke elicits acidosis in brain (
Back et al., 2000
)...
Zhixue Liu
,
et al.
Neuroprotective Actions of PIKE-L by Inhibition of SET Proteolytic Deg...
...This may increase viscosity in the microcirculation and accounts, in part, for the marked acidosis in flow-compromised areas [
7
]...
...Potential underlying causes for lesion growth (studied mainly in focal cerebral ischemia) include: (i) the timedependent increase in microcirculatory flow disturbances [37, 55], (ii) the occurrence of severe lactacidosis in the infarct core [6,
7
], (iii) the occurrence of spreading depression-like phenomena in the periinfarct area [8, 52, 66], (iv) the time-dependent increase in the glucose utilization/blood flow uncoupling in the infarct ...
...The lactate spreads over large parts of the affected hemisphere and (together with hydrogen ions from ATP hydrolysis) contributes to the severe acidosis that may further damage neurons [6,
7
]. Apart from these acute metabolic changes, molecular responses such as the upregulation of immediate-early genes, the formation of stress proteins, the upregulation of pro-apoptotic proteins and inflammatory changes occur that may modulate the final ...
Tobias Back
,
et al.
Lesion evolution in cerebral ischemia
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Citations
(37)
Burning mouth syndrome: A comprehensive review of literature
Anubhav Shivpuri
,
Sunil Sharma
,
Mridula Trehan
,
Nishant Gupta
Journal:
Asian Journal of Oral and Maxillofacial Surgery
, vol. 23, no. 4, pp. 161-166, 2011
Metabolic and perfusion responses to recurrent peri-infarct depolarization during focal ischemia in the Spontaneously Hypertensive Rat: dominant contribution of sporadic CBF decrements to infarct expansion
Yoshimasa Takeda
,
Liang Zhao
,
Michael Jacewicz
,
William A Pulsinelli
,
Thaddeus S Nowak
Journal:
Journal of Cerebral Blood Flow and Metabolism - J CEREBR BLOOD FLOW METABOL
, vol. 31, no. 9, pp. 1863-1873, 2011
Calcium-permeable ion channels involved in glutamate receptor-independent ischemic brain injury
Ming-hua Li
,
Koichi Inoue
,
Hong-fang Si
,
Zhi-gang Xiong
Journal:
Acta Pharmacologica Sinica - ACTA PHARMACOL SIN
, vol. 32, no. 6, pp. 734-740, 2011
ASICs aggravate acidosis-induced injuries during ischemic reperfusion
(
Citations: 1
)
Ling Gu
,
Xiaoyu Liu
,
Yi Yang
,
Dijun Luo
,
Xiaoxiang Zheng
Journal:
Neuroscience Letters - NEUROSCI LETT
, vol. 479, no. 1, pp. 63-68, 2010
Ventilatory patterning in a mouse model of stroke
(
Citations: 1
)
Brian B. Koo
,
Kingman P. Strohl
,
Carl B. Gillombardo
,
Frank J. Jacono
Journal:
Respiratory Physiology & Neurobiology - RESPIR PHYSIOL NEUROBIOL
, vol. 172, no. 3, pp. 129-135, 2010