U50,488H postconditioning reduces apoptosis after myocardial ischemia and reperfusion,10.1016/j.lfs.2010.10.018,Life Sciences,Guang Tong,Zhongchan Sun

U50,488H postconditioning reduces apoptosis after myocardial ischemia and reperfusion (Citations: 1)


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AimsEvidence has indicated U50,488H, a selective κ-opioid receptor (κ-OR) agonist, administered before ischemia attenuates apoptosis and infarction during ischemia and reperfusion (I/R). However, it remains unclear whether U50,488H postconditioning reduces apoptosis during I/R. This study was designed, therefore, to test the hypothesis that U50,488H administered at the onset of reperfusion inhibits cardiomyocyte apoptosis and to investigate the underlying mechanisms.

Journal: Life Sciences - LIFE SCI , vol. 88, no. 1, pp. 31-38, 2011

DOI: 10.1016/j.lfs.2010.10.018

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Keywords (4)

U50,488H postconditioning reduces apoptosis after myocardial ischemia and reperfusion,10.1016/j.lfs.2010.10.018,Life Sciences,Guang Tong,Zhongchan Sun

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Citations (1)

Single Pulse Laser Ablation of Thin Metal Films


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